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Pain ≠ Damage: Understanding Pain and How to Manage It





Pain is something we all experience, yet it remains widely misunderstood. Many people assume that pain directly indicates injury or tissue damage, but modern pain science reveals a more complex reality. While pain can serve as a warning system to protect us, it is also influenced by a complex interaction of biological, psychological, and social factors. Understanding how pain works—and why it doesn’t always mean harm—can help us manage it more effectively. This article explores the science behind pain, the difference between acute and chronic pain, and practical strategies for moving beyond pain to restore function and well-being.


Pain is a vital survival mechanism designed to protect us from harm. It is a complex process involving the nervous system. When the body encounters a potentially harmful stimulus—such as touching something hot or twisting an ankle—specialised nerve endings called nociceptors detect the threat. These nociceptors send electrical signals through the peripheral nerves to the spinal cord, where they are processed and relayed to the brain. However, pain is not just a matter of detecting injury. The brain plays a crucial role in interpreting these signals based on multiple factors, including past experiences, emotions, stress levels, and even environmental context. For example, a soldier in battle may sustain a serious wound but feel little to no pain in the moment because their brain prioritises survival over pain perception. In contrast, a minor paper cut can feel disproportionately painful because the brain deems it significant.


Pain can be acute or chronic. Acute pain occurs as a direct response to tissue injury or inflammation, such as a sprained ankle or a burn. It serves an essential function by encouraging rest and healing. Chronic pain, on the other hand, persists beyond normal tissue healing time—often lasting months or even years. In chronic pain conditions, the nervous system becomes hypersensitive, meaning that pain signals are amplified or even triggered without ongoing tissue damage. This is known as central sensitisation, where the spinal cord and brain adapt in a way that makes the body more responsive to pain signals, even in the absence of an actual threat. Understanding that pain is not always a direct measure of damage is key to managing persistent pain conditions effectively.

 

How Does Chronic Pain Develop?


Chronic pain could be compared to an overactive alarm system—like a car alarm that blares at the slightest touch, even when no real danger exists. Normally, pain is a protective response to injury, alerting us to potential harm. However, in chronic pain conditions, the nervous system becomes hypersensitive, meaning it continues to send pain signals even when there is no ongoing tissue damage. This heightened sensitivity is driven by a process called central sensitisation, where the spinal cord and brain become more efficient at producing and perceiving pain. Instead of pain being a direct reflection of injury, it becomes a persistent experience that can be disproportionate to any actual physical harm.


Several factors contribute to the persistence of pain. One key factor is the fear of movement, also known as kinesiophobia. When someone experiences pain, they may avoid activity out of fear that movement will worsen their condition. However, prolonged avoidance leads to deconditioning—muscles weaken, joints stiffen, and the body becomes even more sensitive to pain. Over time, this creates a cycle where inactivity increases pain levels rather than reducing them. Additionally, emotional and lifestyle factors play a crucial role. Chronic stress, poor sleep, and anxiety can amplify pain perception by altering how the brain processes pain signals. The brain integrates not only sensory input from the body but also psychological and social factors, meaning that chronic pain is a complex interaction between biological and emotional processes.


Understanding that chronic pain is not simply a sign of continued injury, but rather a change in how the nervous system functions, can help people take a more active role in managing their symptoms. Approaches such as gradual exposure to movement, stress management, and improving sleep can help retrain the nervous system and reduce pain over time.

 

Does Pain Always Mean Tissue Damage?





While pain often signals injury, it doesn’t always mean that tissues are damaged. A paper cut is a great example—it can be incredibly painful despite only a minor break in the skin. Conversely, some people have severe arthritis visible on an MRI but report no pain at all. Similarly, studies have found that many individuals with no history of back pain still show disc bulges or degenerative changes on MRI scans, proving that structural changes don’t always correlate with pain.


One reason for this is the nervous system’s sensitivity. When an injury occurs, pain helps protect the body while healing takes place. But in some cases, the nervous system remains in a heightened state of alert, continuing to send pain signals even after tissues have healed (central sensitisation). A striking example of this is phantom limb pain, where individuals experience pain in a limb that has been amputated—proving that pain is generated in the nervous system, not just the injured tissue.


Understanding that pain is influenced by more than just tissue damage allows for a more effective approach to pain management, focusing on movement, lifestyle, and nervous system retraining rather than just structural findings on a scan.

 

 

When Is It Okay to Exercise With Pain?


Experiencing some pain during exercise doesn’t always mean you should stop. In fact, movement is often beneficial—especially for conditions like osteoarthritis, tendinopathy, or persistent pain—as long as the pain is mild to moderate and doesn’t worsen over time. A key principle in pain science is that hurt doesn’t always mean harm. The body can become hypersensitive to pain, and gradually introducing movement can help reduce sensitivity and improve function over time.


For example, people with knee osteoarthritis often feel stiff and sore in the morning, but gentle movement can ease discomfort by improving circulation and joint lubrication. Similarly, in conditions like chronic low back pain, controlled exercise helps retrain the nervous system, reducing pain over time.


A useful guideline is the 0-10 pain scale: If pain stays under 4/10 or improves as you continue moving, it’s generally safe. Another helpful rule is the 24-hour response—if pain settles within 24 hours, the activity was likely appropriate. However, if the pain is sharp, stabbing, worsening, or lingers beyond 24 hours, it may indicate excessive strain and should be modified. Ultimately, the goal is to challenge but not overload the body, allowing adaptation while maintaining safety.

 

When Should You NOT Exercise With Pain?


While movement is often beneficial, certain types of pain may indicate a potential injury or medical condition that requires caution. If pain is sharp, sudden, or worsens significantly during exercise, it could signal a more serious injury. Persistent pain that lasts for several days and is accompanied by swelling, bruising, or loss of function may indicate an underlying issue, such as a muscle tear, ligament injury, or joint inflammation.


Some pain may also be a sign of more serious conditions that require medical attention. For example, unexplained weight loss with ongoing pain could suggest a systemic illness or underlying pathology. Pain that comes with numbness, tingling, or muscle weakness may indicate nerve compression or damage, such as in sciatica or a herniated disc. Additionally, pain following a fall, accident, or direct trauma should be assessed to rule out fractures, ligament tears, or other structural injuries that require medical intervention.


If any of these warning signs are present, it’s best to stop exercising and seek professional advice before continuing with physical activity. Ignoring these symptoms may lead to worsening injury or delayed recovery.

 

How Physiotherapy Can Help You Move Past Pain


Physiotherapy is not just about treating injured tissues—it’s about retraining the nervous system to respond appropriately to movement and sensation. When pain persists beyond normal healing time, the nervous system can become hypersensitive, meaning it sends pain signals even when no actual damage is occurring. This is particularly common in chronic pain conditions like persistent low back pain, osteoarthritis, and post-injury pain syndromes. Physiotherapists play a crucial role in helping the body and brain relearn safe movement patterns and reducing unnecessary pain responses.


A physiotherapist can guide you through graded exposure, a technique that gradually introduces movement to desensitise the nervous system. Instead of avoiding painful activities altogether, controlled and progressive movement helps rewire pain pathways, reducing fear and sensitivity over time. This is particularly useful for conditions where pain persists despite no ongoing tissue damage, such as in central sensitisation syndrome.


Beyond movement, physiotherapy also focuses on education and self-management strategies. Understanding that pain does not always equal harm can empower individuals to move with confidence instead of fear. Stress management techniques, breathing exercises, and cognitive strategies can also help regulate the nervous system and reduce pain intensity. By addressing biological, psychological, and social factors that contribute to pain, physiotherapy goes beyond symptom relief—it restores function and improves overall well-being.


Ultimately, the goal of physiotherapy is not just to reduce pain but to help individuals regain control over their bodies, restore function, and return to the activities they love. By adopting a movement-focused, evidence-based approach, physiotherapists help people move past pain and toward a healthier, more active life.


Conclusion


Pain is a complex and deeply personal experience influenced by factors far beyond tissue damage alone. While acute pain serves a protective function, chronic pain often reflects changes in the nervous system rather than ongoing injury. Recognising this distinction is crucial for effective pain management. Movement, when appropriately guided, can be a powerful tool in breaking the cycle of persistent pain, helping individuals regain confidence and function. Physiotherapy plays a key role in this process, providing evidence-based strategies to retrain the nervous system and promote recovery. By shifting our understanding of pain, we can move beyond fear and take active steps toward a healthier, more resilient body.



 

References


Brinjikji, W., Luetmer, P. H., Comstock, B., Bresnahan, B. W., Chen, L. E., Deyo, R. A., Halabi, S., Turner, J. A., Avins, A. L., James, K., Wald, J. T., Kallmes, D. F., & Jarvik, J. G. (2015). Systematic literature review of imaging features of spinal degeneration in asymptomatic populations. American Journal of Neuroradiology, 36(4), 811–816. https://doi.org/10.3174/ajnr.A4173


Butler, D. S., & Moseley, G. L. (2013). Explain pain (2nd ed.). Noigroup Publications.


Cui, A., Li, H., Wang, D., Zhong, J., Chen, Y., & Lu, H. (2020). Global, regional prevalence, incidence and risk factors of knee osteoarthritis in population-based studies. EClinicalMedicine, 29, 100587. https://doi.org/10.1016/j.eclinm.2020.100587


Dieppe, P. A., & Lohmander, L. S. (2005). Pathogenesis and management of pain in osteoarthritis. The Lancet, 365(9463), 965–973. https://doi.org/10.1016/S0140-6736(05)71086-2


Flor, H. (2002). Phantom-limb pain: Characteristics, causes, and treatment. The Lancet Neurology, 1(3), 182–189. https://doi.org/10.1016/S1474-4422(02)00074-1


Gatchel, R. J., Peng, Y. B., Peters, M. L., Fuchs, P. N., & Turk, D. C. (2007). The biopsychosocial approach to chronic pain: Scientific advances and future directions. Psychological Bulletin, 133(4), 581–624. https://doi.org/10.1037/0033-2909.133.4.581


Henschke, N., Maher, C. G., Refshauge, K. M., Herbert, R. D., Cumming, R. G., Bleasel, J., York, J., Das, A., & McAuley, J. H. (2009). Prognosis in patients with recent onset low back pain in primary care: Inception cohort study. BMJ, 339, b3829. https://doi.org/10.1136/bmj.b3829


Hodges, P. W., & Smeets, R. J. (2015). Interaction between pain, movement, and physical activity: Short-term benefits, long-term consequences, and targets for treatment. Clinical Journal of Pain, 31(2), 97–107. https://doi.org/10.1097/AJP.0000000000000098


Latremoliere, A., & Woolf, C. J. (2009). Central sensitization: A generator of pain hypersensitivity by central neural plasticity. Journal of Pain, 10(9), 895-926. https://doi.org/10.1016/j.jpain.2009.06.012


Lethem, J., Slade, P. D., Troup, J. D. G., & Bentley, G. (1983). Outline of a fear-avoidance model of exaggerated pain perception. Behaviour Research and Therapy, 21(4), 401–408. https://doi.org/10.1016/0005-7967(83)90009-8


Moseley, G. L. (2004). Evidence for a direct relationship between cognitive and physical change during an education intervention in people with chronic low back pain. European Journal of Pain, 8(1), 39-45. https://doi.org/10.1016/S1090-3801(02)00067-1


Nijs, J., Apeldoorn, A., Hallegraeff, H., Clark, J., Smeets, R., Malfliet, A., & Kregel, J. (2019). Low back pain: Guidelines for the clinical classification of predominant neuropathic, nociceptive, or central sensitization pain. Pain Physician, 22(6), 533-546.


Silbernagel, K. G., Thomeé, R., Eriksson, B. I., & Karlsson, J. (2007). Continued sports activity, using a pain-monitoring model, during rehabilitation in patients with Achilles tendinopathy: A randomized controlled study. The American Journal of Sports Medicine, 35(6), 897–906. https://doi.org/10.1177/0363546506298279


Smith, B. E., Hendrick, P., Smith, T. O., Bateman, M., Moffatt, F., Rathleff, M. S., Selfe, J., Logan, P., & Riley, R. (2017). Should exercises be painful in the management of chronic musculoskeletal pain? A systematic review and meta-analysis. British Journal of Sports Medicine, 51(23), 1679–1687. https://doi.org/10.1136/bjsports-2016-097383


Tracey, I., & Bushnell, M. C. (2009). How neuroimaging studies have challenged us to rethink: Is chronic pain a disease? The Journal of Pain, 10(11), 1113–1120. https://doi.org/10.1016/j.jpain.2009.09.001


Tracey, I., & Mantyh, P. W. (2007). The cerebral signature for pain perception and its modulation. Neuron, 55(3), 377–391. https://doi.org/10.1016/j.neuron.2007.07.012


Treede, R. D., Rief, W., Barke, A., Aziz, Q., Bennett, M. I., Benoliel, R., Cohen, M., Evers, S., Finnerup, N. B., First, M. B., Giamberardino, M. A., Kaasa, S., Kosek, E., Lavand’homme, P., Nicholas, M., Perrot, S., Scholz, J., Schug, S., Smith, B. H., … Wang, S. J. (2015). A classification of chronic pain for ICD-11. Pain, 156(6), 1003–1007. https://doi.org/10.1097/j.pain.0000000000000160


Vlaeyen, J. W., & Linton, S. J. (2012). Fear-avoidance model of chronic musculoskeletal pain: 12 years on. Pain, 153(6), 1144-1147. https://doi.org/10.1016/j.pain.2011.12.009


Woolf, C. J. (2011). Central sensitization: Implications for the diagnosis and treatment of pain. Pain, 152(3), S2–S15. https://doi.org/10.1016/j.pain.2010.09.030

 
 
 

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